Voice and Craniofacial Disorders
By: Grace Castillo & Christine Truong
Voice and Craniofacial Disorders By: Grace Castillo & Christine - - PowerPoint PPT Presentation
Voice and Craniofacial Disorders By: Grace Castillo & Christine Truong What is a Voice Disorder? A voice disorder is differences in quality, loudness, and pitch compared to normal. Normal being different than people with
By: Grace Castillo & Christine Truong
“normal”. ○ “Normal” being different than people with similar:
○ Perceptual features (what we hear vs. what we expect to hear) deviate from norms such that they draw attention of those listening
the vocal folds vibrate
sound signal
patients
fold vibrations
muscular tension and effort
glottal level
pitches during phonation
during phonation
affecting the production of fricatives, affricates, and plosives
○ Phonotrauma ■ Repetitive throat clearing ■ Coughing ■ Talking over background noise
○ Direct and indirect surgeries ○ Medical/health conditions ○ Medications
○ Environmental stress ○ Identity conflict ■ Gender dysphoria ○ Psychological and psychiatric problems
○ The primary CN involved in laryngeal innervation
○ Motor: controls most muscles of the larynx, pharynx, and palate for phonation, swallowing, and resonance; controls the gag reflex with CN IX ○ Sensory: sensation from posterior tongue and larynx; regulates oxygen intake and lung Inflation
○ Internal branch provides all of the sensory information to the larynx ○ External branch supplies motor innervation solely to the cricothyroid muscle
○ Supplies all motor innervation to the interarytenoid, posterior cricoarytenoid, thyroarytenoid, and lateral cricoarytenoid muscles ○ Supplies all sensory information below the vocal folds
○ Innervates the posterior belly of the digastric muscle of the vocal folds
○ One of the suppliers for the power of the voice
○ Air movement (transfers air in/out of the lungs)
○ Gas movement across membranes ○ Exchanging: ■ Atmospheric gas with that of blood gases ■ Blood gases with that or organ-producing gases ■ Take oxygen in and release carbon dioxide
and no deeper
○ Raising the clavicle & shoulders during Inhalation ○ Keep the rest of the torso motionless
○ The diaphragm is a dome-shaped respiratory muscle found near the bottom of your ribcage. When you inhale and exhale air, the diaphragm & other respiratory muscles around your lungs contract. ○ During inhalation, your diaphragm contracts so that your lungs can expand into the extra space and let in as much air as is necessary. ○ Muscles in between your ribs, known as intercostal muscles, raise your rib cage in order to help your diaphragm let enough air into your lungs.
○ Start by inhaling (this expands the thorax) ○ This causes the intercostal muscles to elevate the ribs (as compared to abdominal breathing using the diaphragm)
Myo = Muscle Elastic = Stretchy Aerodynamic = Airflow & pressure
interact with VF tissues to set them in motion for a time
they contribute to VF movement
○ Horizontally: closed at midline to opening out (clap hands) ○ Vertically: down to up (zipper) ○ Longitudinally: front to back
types of movements
○ Air from lungs moves in one direction ○ Air pressure builds up below the VFs, pulls them apart, and then the VFs are pulled back together ○ As the VFs move closer together, airflow between them is reduced (less space for air to pass) ○ Air above closing VFs continues to move up at the same speed (inertia) ○ Speed of air pressure is maintained except for just above the VFs
○ Added 2 other masses: VF cover & thyroarytenoid ○ Masses move independently yet are linked ○ At different points in the vibration cycle ■ Bottom portion of the VFs are farther apart than the upper portion and vice versa ○ There is greater air pressure when bottom parts are further apart ■ Uneven air pressure maintains VF vibration
pressure
resonating cavities
○ Oral cavity ○ Nasal cavity
○
Pharynx
○ (1) Epithelium ○ Lamina Propria ■ (2) Superficial layer (Reinke’s Space) ■ (3) Intermediate layer + (4) Deep layer = Vocal Ligament ○ (5) Vocalis Muscle [Thyroarytenoid muscle]
○ Fundamental frequency (FF) - rate of vibrations of the VFs ○ Measured in Hz ○ Phonatory modes or registers ■ Falsetto ■ Modal ■ Glottal fry ○ Factors affecting FF ■ VF length ■ VF tension ■ Subglottic pressure ■ Amplitude of vibration
○ Perceptual judgments of quality are often subjective, though standard measures make this somewhat more objective ○ Factors that influence phonatory quality: ■ Integrity of vibration: regular, symmetry, phase shape/closure and slope of glottal flow waveform ■ Integrity of the respiratory system ■ Supraglottic vocal tract
○ Vocal intensity - SPL of acoustic signal we hear ○ Measured in dB ○ Factors affecting intensity: ■ Subglottic air pressure ■ Transglottic air flow ■ VF vibration phase closure ■ Supraglottic vocal tract resonating cavities
○ Structural ○ Inflammatory ○ Trauma/injury ○ Systemic ○ Nonlaryngeal Aerodigestive ○ Psychiatric/Psychological ○ Neurologic ○ Other Disorders ■ Separate Category: Gender Dysphonia & Gender Reassignment
Etiology Characteristics Treatment Vocal Nodules
difficulty breathing
proper voice use
Vocal Polyps
Vocal Cysts
Does not respond to therapy!!!
Reactive VF Lesions
VF cyst; it’s a reaction to a unilateral VF lesion
Etiology Characteristics Treatment Reinke’s Edema
VF Scarring
abuse
permanent damage Sulcus Vocalis
Granuloma
from irritated vocal tissue
changes
Etiology Characteristics Treatment Fibrous Mass
material within the lamina propria
Pseudocyst
Contact Ulcer
tube)
reflux)
Subglottic and Glottic Stenosis and Acquired Anterior Web
trauma (GSW)
breaths, stridor (high pitched sound during breathing)
Etiology Characteristics Treatment VF Hemorrhage, Hematoma, Varix, Extasia
medicines
and roughness
affected
bleeding (steroids)
days (no coughing, no talking, no throat clearing) Epithelial hyperplasia aka keratosis
Leukoplakia
Erythroplasia
voice use and chemical irritants
Etiology Characteristics Treatment Recurrent Respiratory Papilloma
Epithelial dysplasia
alcohol
Laryngectomy
lack of a larynx
Puberphonia (Falsetto, Juvenile)
voice (For males: voice is high. For females: voice is low)
Etiology Characteristics Treatment Congenital Webs
dysphonia
Laryngomalacia
Presbylaryngis/ Presbyphonia
quality
and flexibility
therapy
Etiology Characteristics Treatment Cricoarytenoid & Cricothyroid Arthritis Laryngitis
corticosteroid medications Reflux (GERD & LPR)
pharynx and larynx
Irritable Larynx Syndrome (ILS)
Etiology Characteristics Treatment Thermal and Chemical Exposure
chemicals or heat
Intubation/Extubation Injury
strategies Arytenoid Dislocation
penetrating wound injury
Etiology Characteristics Treatment Hypothyroidism
Hyperthyroidism
Sexual Hormone Imbalances
levels
Allergies
Rheumatoid Arthritis (Also Inflammatory)
Etiology Characteristics Treatment Asthma
COPD
Reflux (GERD)
pharynx and larynx
Croup
Candida
quality
Etiology Characteristics Treatment Functional Dysphonia/Aphonia
imbalance; there is no visual structural or neurological pathology
loss of voice
Malingering
pretending/inventing
gain
malingerer
Etiology Characteristics Treatment PNS: VF Paresis/Paralysis
the PNS commonly caused as a side effect of surgeries
breathiness, fatigue, effortful phonation, aphonia
Movement disorder: Spasmodic Dysphonia
but research has shown that damage of the basal ganglia causes this
adduction and abduction of the VFs during phonation, laughing, crying, coughing, etc.
Etiology Characteristics Treatment Vocal Abuse/Misuse
behaviors that have a negative impact
Muscle Tension Dysphonia
cause *our voice book says functional dysphonia is a cause*
compensate either for a laryngeal pathology, paralysis,
Ventricular Phonation (Plica Ventricularis)
when it shouldn’t be
Etiology Characteristics Treatment Vocal Fatigue
pharyngeal muscles, increased thickness of the first two layers, reduced internal hydration, reduced respiratory pressure
Paradoxal VF Motion or VF Dysfunction
abnormally move and move together unexpectedly
Irritable Larynx Syndrome
Etiology Characteristics Treatment Gender Reassignment
resonance, intonation, rate, and/or volume
pitch modification Gender Dysphonia
associated with the identification of a different gender
Glottic Stenosis VF Polyp Reinke’s Edema VF Hemorrhage; Vascular Lesion Recurrent Respiratory Papilloma Pseudocyst
VF Nodules Fungal Infection VF Cyst Granuloma
○ Esophageal speech ■ Patients literally speak on burps or belches ■ Produced by the pharyngo-esophageus which is the vibratory source ○ Artificial larynx (electrolarynx) ■ A mechanical, handheld device that generates sound ○ Tracheoesophageal speech (TEP)
■
A prosthetic device that is inserted into a tracheoesophageal wall puncture
○ Determine etiology ○ Describe the problem ○ Evaluate how the disorder affects vocal components ○ Generate the treatment plan
referral source
○ Nature of voice disorder ○ Onset, course, and duration ○ Risk factors ○ Medical issues and medications ○ Social history ○ Voice use ○ Hydration
○ CN exam (include a detailed
○ Auditory-perceptual eval (clinical voice eval) ■ Respiration and posture, phonation/quality, loudness, pitch ○ Use various voice measures and severity rating scales (e.g. CAPE-V, Mayo Severity Rating Scale, GRBAS)
○ Acoustic measures - pitch, loudness, etc. (e.g. Visipitch) ○ Laryngeal imaging (endoscopy and videostroboscopy use various tasks and measures)
○ VHI-10 ○ CSI ○ RSI ○ SVHI-10
○ Prognosis
○ Treatment methods
○
Long term goals
○
Short term goals
○
Duration and frequency of therapy
instrumentation and some kind of light source
structures/functions with an unaided eye
through the oral cavity
through the nasal cavity to visualize the VFs
visualizing the VFs
to visualize VF vibration in what appears to be slow motion
parameters which include the following:
○ Pitch ○ Loudness ○ Resonance ○ Respiration ○ Phonation
assessments
○ A way to help bring voice change to a level that can be achieved and to achieve realistic voice change to meet the maximum extent of needs as much as possible
○ Reduce/remove barriers to change ○ Provide Feedback ○ Be aware of emotional distress ○ Tailor therapy to individual needs
involves reducing/removing barriers to change
○ Multiple factors affect this: ■ Willingness to change, how they responded to change in the past, whether they have family support, their beliefs/belief system
the day and/or duration of therapy
used to establish a baseline before starting therapy but also for demonstrating progress, change, and desired goals
○ Daily life and associated problems, self-concept, etc.
sometimes the cause may be unknown, or the cause may not be able to be changed
○ It may not be within the SLP scope of practice to change it
○ Voice needs ○ Gender ○ Age ○ Health needs ○ Awareness of the problem ○ Motivation to change ○ Culture ○ Intelligence/education ○ Occupation ○ Family support
○ Hygienic/vocal hygiene ○ Symptomatic ○ Physiologic ○ Psychologic
○ Hygienic/vocal hygiene ■ Negative behavior leads to the voice problem ■ Reduce /eliminate negative behavior to improve/resolve the voice disorder
○ Identify misuse during assessment ○ Reduce /eliminate through various voice techniques
○ Disruption of normal physiologic balance of vocal mechanism can cause a voice disorder
○ While the vocal structure is normal, something has changed within the pt that is manifested in his/her voice
○ Refers to an abnormality of the face and/or head ○ Can be mild or severe ○ A congenital condition ○ Can be an isolated abnormality or associated with a sequence or syndrome
○ Occurrence of 2 or more anomalies in a single individual where all anomalies are caused by a single primary agent ○ Example: Treacher Collins Syndrome or Stickler Syndrome
○ Multiple anomaly disorder where many of the abnormalities are actually secondary disorders ○ Example: Pierre Robin Sequence
triangular area of the hard palate anterior to the incisive foramen and includes a portion of the alveolar ridge
consists of the remaining hard palate and all of the soft palate
○ Velum (soft palate) ○ Lateral pharyngeal walls ○ Posterior pharyngeal wall
that takes place in the nasopharynx and involves movements of the soft palate and pharyngeal walls
airflow in the oral and nasal cavities during speech
anatomy (structure) and normal physiology (movement)
○ If any of these components are disrupted, there is velopharyngeal dysfunction
pre-linguistic and linguistic development
without cleft palate but may have delayed onset of canonical babbling
produced
during babbling
productions
glottal fricative /h/ (compared with typical preference for alveolar stop /d/)
words more slowly
beginning with sonorants (nasals, liquids, glides, vowels)
○ Articulation may be characterized by a restricted phonetic inventory, sound substitutions/omissions, and obligatory vs. compensatory articulation errors
with clefting. It depends on the type & severity of the cleft.
pharyngeal swallowing function; once the milk reaches the oropharynx, the swallow is initiated with normal airway protection.
feeding difficulty. Once the nipple is positioned in the baby's mouth, he or she can usually achieve sufficient compression of the nipple against the intact palate.
may have more significant feeding difficulty. They are unable to separate the nasal cavity from the
negative pressure necessary for sucking. In addition, they may have difficulty compressing the nipple to express milk because the palatal surface is not intact.
difficulties include
expended during feeding
nutritional intake
1. What are the 2 cranial nerve branches related to voice?
a. RLN and SLN
2. What are the 3 types of breathing related to voice?
a. Clavicular, Diaphragmatic-Abdominal, and Thoracic
3. Briefly explain the Myoelastic-Aerodynamic Theory
a. Myo=Muscle. Elastic=Stretchy.Aerodynamic=Airflow & pressure. Problem: VF vibration will dampen over time
4. Briefly explain Hirano’s Body-Cover Theory.
a. VFs can move in 3 ways: horizontally, vertically, longitudinally
5. Briefly explain Titze’s Self-Oscillation Theory.
a. One mass: Air pressure builds up below the VFs, pulls them apart, and then the VFs are pulled back together. It moves in 1 direction b. 3 mass: Masses move independently yet are linked
6. What are the 5 layers of the VF?
a. Epithelium, Lamina Propria (Superficial, Intermediate, Deep), Vocalis
7. Briefly explain the etiology, characteristics, and treatment for nodules.
a. Etiology: phonotrauma, vocal misuse, vocal abuse b. Characteristics: rough, breathy, tense, difficulty breathing c. Treatment: voice therapy to ensure proper voice use, surgery
8. What are the 3 voice characteristics?
a. Quality, pitch, loudness
9. What are the 4 treatment approaches?
a. Hygienic, Symptomatic, Physiologic, Psychologic
10. Name the 4 general principles of voice therapy.
a. Reduce/remove barriers to change, provide feedback, be aware of emotional distress, tailor therapy to individual needs