Bioidentical Hormone Therapy: Hormone Therapy? What? Why? How? Why - - PDF document

bioidentical hormone therapy
SMART_READER_LITE
LIVE PREVIEW

Bioidentical Hormone Therapy: Hormone Therapy? What? Why? How? Why - - PDF document

Bioidentical Hormones: What Are the Issues? QUESTIONS NAMS Annual Meeting NAMS Annual Meeting October 12, 2013 What IS Bioidentical (or Human identical) Bioidentical Hormone Therapy: Hormone Therapy? What? Why? How? Why is


slide-1
SLIDE 1

Bioidentical Hormones: What Are the Issues? NAMS Annual Meeting NAMS Annual Meeting October 12, 2013

Bioidentical Hormone Therapy: What? Why? How?

Jan L. Shifren, M. D.

Associate Professor of Obstetrics, Gynecology and Reproductive Biology Harvard Medical School Director, Menopause Program, Vincent Ob/Gyn Service Division of Reproductive Endocrinology and Infertility Massachusetts General Hospital

What IS “Bioidentical” (or “Human identical”) Hormone Therapy? Why is Bioidentical Hormone Therapy

QUESTIONS

Controversial? Why Consider BioHT vs. “Traditional” HT? How May BioHT Be Prescribed consistent with high standard of evidence-based care?

50-59 60-69 70-79 <10 10-19 >20 Years Since Outcome Age (years) Menopause Absolute Excess Risks (cases per 10,000 person/years) by Age and Years Since Menopause in Combined WHI Trials (E+P and E-Alone) CHD

  • 2
  • 1

+19*

  • 6

+4 +17* Total mortality -10

  • 4

+16*

  • 7
  • 1

+14 Global index†

  • 4

+15 +43 +5 +20 +23

* P=0.03 compared with age 50-59 years or <10 years since menopause

Global index is a composite outcome of CHD, stroke, pulmonary embolism, breast cancer colorectal cancer, endometrial cancer, hip fracture and mortality Roussouw JE, et al. JAMA 2007;297:1465

HT should be prescribed at “lowest effective dose”, guided by symptoms Serum hormone levels in reproductive aged women vary throughout day and menstrual cycle, so

Assessing Hormone Levels to Adjust “BioHT” Dosing Provides False Sense of Efficacy & Safety

y g y y , impossible to “match” an individual woman’s “ideal” hormone levels Checking serum levels with compounded HT products likely necessary for safety, not efficacy, due to risk of excessive dosing Salivary hormone levels not reproducible, poorly reflect serum levels, affected by diet and other variables Luteal phase levels: ~ 5-16 ng/ml Patch size required: ~ 30 times E2 patch Study of 64 mg P-cream: slight increase circulating P levels, insufficient to achieve any

Transdermal Progesterone Cream: Insufficient to Induce a Detectable Effect on Endometrium!!

biological response in endometrium DBRCT P-cream (32 mg QD) vs. Placebo x 12 weeks No change in VMS, mood, sexuality, serum lipids, bone markers Progesterone levels increased from 0.1 to 0.3 ng/ml

Wren BG et al. Menopause 2003; 10:13 Gambrell RD. Menopause 2003; 10:1-3 Wren BG et al. Climacteric 2000; 3:155

Oral vs. Transdermal Estrogen Delivery Transdermal E2 does not undergo first-pass hepatic metabolism

Transdermal Bloodstream Oral Stomach Intestines Liver Liver proteins

Cells of target tissues

Hemelaar M, et al. Fertil Steril 2008:90:642 Filer WD, Filer RB. Am Fam Physician 1994

HDL, LDL, Triglycerides Renin substrate Antithrombin III C-reactive protein (CRP) Activated protein C resistance (APCr)

slide-2
SLIDE 2

Hormone Therapy and Venous Thromboembolism Among Postmenopausal Women

Current Estrogen Therapy VTE Cases (DVT & PE)

(n = 259)

Controls

(n = 603)

Risk VTE compared to non-users

(Odds Ratio

Canonico M, et al. Circulation 2007; 115:840

with 95% CI)

Oral Estrogen 45 39 4.2 (1.5-11.6) Transdermal Estrogen 67 180 0.9 (0.4-2.1) Compared with HT Non-Use:

Risk of Stroke: Transdermal vs. Oral Hormone Therapy

Population based nested case-control study United Kingdom’s General Practice Research Database Women aged 50-79 years 15,710 cases stroke matched to 59,958 controls

p Transdermal HT: RR 0.95 (0.75-1.2) Low dose E2 patches: RR 0.81 (0.62-1.05) High dose E2 patches: RR 1.89 (1.15-3.11)* Oral HT (low & high dose): RR 1.69 (1.15-1.42) *

Renoux C et al. BMJ 2010; 340:2519

RR: rate ratio Low dose E2 patches: <50 mcg Low dose oral E: CE <0.625 or E2 <2 mg

Benefits

Choice of formulation (patch, gel, spray) Convenience (1-2 patches weekly) Consistent blood levels Multiple & low doses “Bioidentical estradiol”

  • Transdermal vs. Oral Hormone Therapy

No ‘first pass hepatic effect’ (limited effect on lipids, clotting factors, free testosterone, thyroid) Possible lower risk VTEs & CVA

Drawbacks

Cost Limited E + P combination products Topical gels less convenient (daily dosing) Patch irritation

Effects of Estrogen or Estrogen/Progestin Regimens on Heart Disease Risk Factors in Postmenopausal Women: PEPI Trial

Lipid Parameter (mg/dl) Placebo Estrogen (CE) Alone Estrogen (CE) + MPA (cyclic) Estrogen (CE) + Progesterone (cyclic)

Changes from Baseline Lipid Values

Writing Group PEPI Trial. JAMA 1995; 273:199

HDL

  • 1.2

+ 5.6*9 + 1.6* + 4.1*9

LDL

  • 4.1
  • 14.5*
  • 17.7*
  • 14.8*

Triglycerides

  • 3.2

+ 13.7* + 12.7* + 13.4*

Total Cholesterol

  • 4.2
  • 7.6
  • 14.1*
  • 7.8

*p <0.001 vs. placebo 9 p<0.004 vs. regimens w/ MPA

Compared with HT Never-Use: Estrogen alone: RR 1 29 (1 02 1 65)*

Breast Cancer Risk Associated with Different Hormone Therapy Regimens

E3N French epidemiologic cohort study Self-administered questionnaires 1990-2002 80,377 postmenopausal women w/ up to 12 years F/U

Estrogen alone: RR 1.29 (1.02-1.65) Estrogen+ progesterone: RR 1 (0.83-1.22) Estrogen + dydrogesterone: RR 1.16 (0.94-1.43) Estrogen + other progestagens: RR 1.69 (1.50-1.91)*

No association with risk according to route of estrogen administration (oral or transdermal)

Fournier A et al. Breast Cancer Res Treat 2008 ; 107:103

ESTRADIOL Systemic doses of estradiol for treatment of hot flashes Oral tablet: Estrace, generics Skin patch: Alora, Climara, Esclim, Menostar, Vivelle Dot Estraderm, generics Skin gel/cream: EstroGel, Elestrin, Divigel, Estrasorb Skin spray: Evamist Vaginal ring Femring

FDA Approved Formulations of “Bio-HT”

Vaginal ring: Femring PROGESTERONE Systemic doses of progesterone to protect endometrium Oral tablet: Prometrium

slide-3
SLIDE 3

Low doses of estradiol for treatment of vaginal dryness and dyspareunia Vaginal cream: Estrace vaginal cream Vaginal ring: Estring Vaginal tablet: Vagifem

FDA Approved Low Dose Vaginal Formulations of “Bio-HT”

Vaginal tablet: Vagifem

Serum Estradiol Levels with Vaginal Estradiol (10 mcg) Tablet Use

13.2 13.2± ±34.4 34.4 11.8 11.8± ±17.59 17.59 14.7 14.7± ±34.4 34.4 13.0 13.0± ±17.59 17.59

g g/mL /mL

Bachmann G, et al. Bachmann G, et al. Obstet Obstet Gynecol

  • Gynecol. 2008;111:67

. 2008;111:67-

  • 76.

76.

Mean E2 Mean E2 pg pg Baseline Baseline Week 52 Week 52

Conclusions: Bioidentical Hormone Therapy

HT is most effective treatment for bothersome VMS & benefits generally outweigh risks for healthy, symptomatic women at menopause transition “Bioidentical HT” (estradiol & progesterone) available with FDA approved products Consider transdermal estradiol option Low dose topical FDA approved “bioidentical” estradiol products available for vaginal dryness & dyspareunia NO benefit & significant potential risk with use of compounded HT

The Naked Truth About Bioidentical Hormones

OR

Bioidentical Hormone Therapy: Custom-Compounded

  • vs. Government-

Approved