Urinary Continence & Management Post Stroke Incontinence and - - PowerPoint PPT Presentation

urinary continence amp management post stroke
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Urinary Continence & Management Post Stroke Incontinence and - - PowerPoint PPT Presentation

Urinary Continence & Management Post Stroke Incontinence and Stroke occurs in greater than 50% of acute stroke patients despite the personal, economic and psychosocial impact treatment evidence specific to stroke remains limited


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Urinary Continence & Management Post Stroke

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Incontinence and Stroke

 occurs in greater than 50% of acute stroke

patients

 despite the personal, economic and

psychosocial impact treatment evidence specific to stroke remains limited

Incontinence is an indicator of stroke severity because of its association with poor outcome, and increased incidence of depression for both survivor and care giver

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Responses to Incontinence

 Fear  Embarrassment  Shame  Anxiety  Frustration  Guilt  Anger

Quality of life can be compromised !

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Contributing Factors Post Stroke

 Motor impairment  Altered LOC  Sensory lesions  Ataxia  Depression  Aphasia  Pre stroke continence issues

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Detrusor muscle relaxes + Urethral Sphincter tone + Pelvic floor tone Detrusor muscle contracts + Urethral Sphincter Relaxes

(Voluntary control)

+ Pelvic floor Relaxes MICTURITION Detrusor muscle relaxed + Urethral Sphincter contracts + Pelvic floor contracts Detrusor muscle relaxes + Urethral Sphincter tone + Pelvic floor tone Bladder filling Bladder filling First sensation to void Normal desire to void Emptying phase Bladder pressure Storage phase

Normal Micturition Cycle

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CNS Control of Bladder

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CNS Control of Bladder Function

Pontine Centre (Pons–brainstem):

 Receives input relayed from the sacral centre  Coordinates detrusor contraction and urethral sphincter

relaxation

 Also communicates with cerebral cortex – voluntary control

Frontal lobes:

 Inhibits detrusor muscle contractions  Overrides the sacral reflex arc and keeps the urethral

sphincter closed

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Normal Bladder Function

Reflexive (involuntary) response

Bladder constantly filling under low pressure

Stretch receptors in bladder wall activated

Impulse sent to the sacral bladder centre (S2-S4)

Causes reflex: Detrusor muscle contracts and internal sphincter relaxes

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Normal Bladder Function

Voluntary control…

 Cerebral cortex (frontal lobe) able to reduce urge and

delay urination

 Inhibits the sacral reflex arc - controls urethral

sphincter (external)

 Pontine micturition center (brainstem) - coordinates

relaxation of external sphincter and detrusor muscle contractions

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Bladder Function: Storage and Voiding

 400-600 ml maximum bladder capacity  150 - 300 ml first desire to void  “Normal” voiding frequency 4-8 times per

day and once at night

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Strokes and Continence

Frontal Stroke

 May maintain voluntary control of the external

sphincter but uninhibited bladder contraction

 Strong urge to void with short or no

warning

 Persistent frequency, nocturia,

urge incontinence

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Parietal Lobe & Basal Ganglion Stroke

 Uninhibited bladder (detrusor) contractions  Urethral sphincter fails to relax  Voiding obstructed - Overflow incontinence  May lead to ureter reflux and renal damage

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Types of Incontinence

+ Functional

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Common Voiding Dysfunctions Post Stroke

 Frequency  Urgency  Urge incontinence  Retention

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Overflow Incontinence

 Bladder constantly full & urine leaks  Related to partial obstruction of

bladder neck (prolapse, BPH) or bladder muscles may be inactive

 Results in dribbling, urgency,

frequency or difficulty initiating stream

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Urge Incontinence

 Overactive detrusor results in

strong urge to void

 Unable to make it to bathroom  Loss of mod amount of urine  Men & women affected  Common in stroke patients

  • 1. Bladder Muscle Contracting
  • 2. Urethral sphincter relaxed
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Functional Incontinence

 UI that results from barriers (functional or

environmental) in reaching the BR in time

 Involves impaired cognitive functioning or

impaired physical functioning

 May be associated with inability to

communicate need to go to B/R

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Assessment of Incontinence

 Incontinence Hx  Past Medical Hx  Functional ability  Medications  Fluid intake

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Assessment: Risk factors of UI

 Caffeine intake  Current UTI  Constipation  Weak pelvic floor muscles  Hx Diabetes

 Mobility issues  Diminished cognitive status  Environmental barriers  Medications e.g. diuretics,

sedatives

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Reversible Causes Incontinence

 D - Delirium  I - Infection  A - Atrophy  P - Pharmacotherapeutics  P – Psychological Issues (Depression)  E – Endocrine issues (High glucose)  R – Restricted Mobility  S – Stool Impaction

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Managing Incontinence

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Baseline Post Void Residuals(PVR)

 Patient voids (measure) & complete PVR (using bladder scan)  If PVRs are less than 150ml over 3 consecutive scans in

72hrs, PVR may be discontinued

 If PVRs = or > 150ml, for 3 consecutive voids over

72hrs, patient has urinary retention

 If patient has both high PVRs and incontinent episodes,

they have overflow incontinence

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Voiding Record

Record: Time and amount x 3-4 days

 fluid intake  urine voided  incontinence (time

& volume)

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Fluid Intake Changes

 Reduce/eliminate caffeine/alcohol/citrus juices, &

artificial sweeteners

 Ensure daily fluid intake

adequate (1500-2000 ml)

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Behavioral Interventions

Use first line before pharmaceuticals or surgery

 Pelvic floor retraining (Kegal exercises)  Prompted voiding  Bladder retraining  Environmental and clothing modifications

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Urge Suppression Strategies

 Prompted voiding initially q 3 h (urge & stress)  Urge suppression using distraction and

relaxation techniques

 Bladder retraining - goal: gradually  voiding intervals

while  voiding volumes (urge)

 Combine with Kegel exercises (mixed)

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Bladder Retraining

After 3-4 days of keeping a voiding record (VR):

Find average time interval between voids

Schedule BR routine 15-30 minutes after average interval noted

  • n VR

If the urge is intense try: deep breath/relax, a few Kegal exercises, count backwards

Try to get to the next scheduled BR visit

Gradually increase the time between BR visits

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Overflow Incontinence

 Post void residual volumes (scanner), normal is

50-100 ml

 Double voiding encouraged  Intermittent catheterization for PVR > 150ml

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Incontinence Products

 Use pads made for urine loss rather than

using menstrual pads, facecloths or tissue

 Use unscented, mild soap sparingly  Local estrogen cream (prescription)

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Pharmacologic Treatment

 Anticholinergic medications  Estrogen

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Anticholinergics:

 Reduce irritability of the bladder, decreasing

uninhibited detrusor muscle contractions

 Allow for larger bladder volumes  Reduces frequency  Suitable for urge incontinence in stroke  e.g. Oxybutinin (Ditropan), Tolterodine (Detrol),

Imipramine (Tofranil)

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Anticholinergics

Side effects:

 dry mouth  drowsiness, fatigue  altered mentation with diminished ability for

complex problem solving

 hypertension, tachycardia  insomnia

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Bowel Management

Constipation & Bowel Incontinence

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Indicator of functional recovery

 Affects quality of life both patient and family  Research demonstrates that improved bowel

function can improve participation in rehab and mobility (Yi, et al, 2011)

 Fecal and urinary incontinence is the second most

common reason for elder institutionalization (Arnold-Long,

2010)

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Constipation and stroke

 Limited research  Common 30-60% of post stroke patients  Caused by limited mobility, lethargy, reduced

fluid intake, depression, cognitive impairment, reduced LOC, meds

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Bowel Incontinence

 Stroke research lacking  Can affect up to 30% of acute stroke patients

and then 11% at 3-12 months post stroke

 Lack of awareness due to infarct  Communication issues  Fecal impaction the primary

cause of fecal incontinence

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Assessment Required

 Hx pre stroke

 constipation, incontinence  hard stools, straining, sensation of incomplete bowel

emptying

 Post Stroke

 toilet access, dependence level including arm fn  inactivity, adequate fluid and food intake, meds

 Rectal exam

 weak pelvic floor or sphincters (anal wink)  impaction

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Defecation Process

 Gastro colic reflex strongest with first meal of day  Internal sphincter- smooth muscle (involuntary)  External sphincter - striated muscle (voluntary)

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Establish a bowel program

 Start with a clean bowel (LES)  Evaluate medications that promote or inhibit bowel function  Encourage appropriate fluids, diet (high fiber), & activity (bed,

chair, ambulating)

 Choose an appropriate rectal stimulant (supp) not laxatives  Provide rectal stimulation initially to trigger defecation daily  Select optimal scheduling (pc first meal) and positioning (toilet,

squat position, knees higher than hips) with feet supported

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Post Stroke Complications Assessments Required !!!

 Initial and ongoing assessments in post stroke

patients can identify complications early on, leading to early intervention and improved

  • utcomes

 This is one of the reasons stroke units have

been proven to reduce morbidity and mortality by 30%

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References

Arnold-Long, M. (2010). Fecal incontinence. Long Term Living. http://www.ltlmagazine.com/article/fecal- incontinence

Brittain, K. (1999). Prevalence and management of urinary incontinence in stroke survivors. Age and Ageing, 28

Canadian Continence Foundation (2007). Incontinence: A Canadian perspective. Retrieved December 2008 from

http://www.canadiancontinence.ca/health-profs/health-profs.html

Harari, D., Norton, C., Lockwood, L., & Swift, C. (2004). Treatment of constipation and fecal incontinence in stroke

  • patients. https://stroke.ahajournals.org/content/35/11/2549.full

Heart and Stroke Foundation (2007) Faaast FAQs for Stroke Nurses

Lindsay MP, Gubitz G, Bayley M, Hill MD, Davies-Schinkel C, Singh S, and Phillips S. Canadian Best Practice Recommendations for Stroke Care (Update 2010). On behalf of the Canadian Stroke Strategy Best Practices and Standards Writing Group. 2010; Ottawa, Ontario Canada: Canadian Stroke Network. http://www.strokebestpractices.ca/index.php/acute-stroke-management/inpatient-management-and-prevention-of- complications-following-acute-stroke-or-tia/

Teasell, R., Foley, N., Salter, K., Bhogal, S. (2008). Medical Complications Post Stroke. Retrieved from www.ebrsr.com

Yi, J., Chun, M., Kim, B., Han, E., & Park, J. (2011). Bowel function in acute stroke. Annals of Rehabilitation Medicine, 2011. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3309224